Your privacy, your choice

We use essential cookies to make sure the site can function. We also use optional cookies for advertising, personalisation of content, usage analysis, and social media.

By accepting optional cookies, you consent to the processing of your personal data - including transfers to third parties. Some third parties are outside of the European Economic Area, with varying standards of data protection.

See our privacy policy for more information on the use of your personal data.

for further information and to change your choices.

Skip to main content
Figure 3 | Molecular Pain

Figure 3

From: p300 exerts an epigenetic role in chronic neuropathic pain through its acetyltransferase activity in rats following chronic constriction injury (CCI)

Figure 3

The effects of LV-shp300 and C646 on p300 expression in the lumbar spinal cord in rats . A: Expression of mRNAs for p300 in the lumbar spinal cord in all rats. The β-actin fragment was used as an internal control. B: The mean (±SEM) of integral optical density (IOD) of P300/β-actin in rats on mRNA level, n = 5. C: Expression of protein p300 in the lumbar spinal cord in all rats. D: The mean (±SEM) of IOD of P300/PCNA in all rats on protein level, n = 5. S: sham-operated rats; NS: NS-treated CCI rats; NC: LV-NC-treated CCI rats; shp300: LV-shp300-treated CCI rats. C37: C37-treated CCI rats; C646:C646-treated CCI rats. *P < 0.05 vs. sham-operated group; #P < 0.05 vs. NS and NC-treated groups.

Back to article page